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Semaglutide: difference between revisions

Diff·revision 13 → 14·23:35, 15 Nov 2024

Difference between revision 13 and revision 14 of Semaglutide. 23 lines changed; the page grew by 1,050 bytes.

Revision 13 — 10:04, 30 Oct 2024
OrforglipronOona (talk)
convert the substitution list to a table so the analogues line up
5,608 bytes +30
Revision 14 — 23:35, 15 Nov 2024
ColdChainCleo (talk)
expand §Chemistry and engineering
6,658 bytes +1,050
12| Molecular formula = {{math|C_{187}H_{291}N_{45}O_{59}}}12| Molecular formula = {{math|C_{187}H_{291}N_{45}O_{59}}}
13| Molar mass = 4,113.58 g·mol⁻¹13| Molar mass = 4,113.58 g·mol⁻¹
+14<!-- Pharmacokinetics -->
+15| Half-life = ≈165 h (7 days)
+16| Bioavailability, SC = ≈89%
+17| Albumin binding = >99%
+18| Time to steady state = 4–5 weeks
14}}19}}
15{{hatnote|For the oral formulation and its absorption enhancer, see [[Oral semaglutide]]. For the drug class, see [[GLP-1 receptor agonist]].}}20{{hatnote|For the oral formulation and its absorption enhancer, see [[Oral semaglutide]]. For the drug class, see [[GLP-1 receptor agonist]].}}
35The oral formulation is a different pharmacokinetic proposition entirely. Bioavailability is roughly 0.4–1%, achieved by co-formulation with the absorption enhancer sodium N-(8-(2-hydroxybenzoyl)amino)caprylate, and it is critically dependent on dosing in the fasting state with no more than 120 mL of water and a subsequent 30-minute fast. Deviation from those conditions changes exposure severalfold.{{r|knudsen2019}}40The oral formulation is a different pharmacokinetic proposition entirely. Bioavailability is roughly 0.4–1%, achieved by co-formulation with the absorption enhancer sodium N-(8-(2-hydroxybenzoyl)amino)caprylate, and it is critically dependent on dosing in the fasting state with no more than 120 mL of water and a subsequent 30-minute fast. Deviation from those conditions changes exposure severalfold.{{r|knudsen2019}}
3641
+42== Clinical evidence ==
+43| Trial | Population | Dose | Primary result |
+44|---|---|---|---|
+45| SUSTAIN 6 | T2D, high CV risk | 0.5/1.0 mg weekly | MACE HR 0.74 (95% CI 0.58–0.95) |
+46| STEP 1 | Obesity, no diabetes | 2.4 mg weekly | −14.9% vs −2.4% weight at 68 wk |
+47| STEP 2 | Obesity with T2D | 2.4 mg weekly | −9.6% vs −3.4% weight at 68 wk |
+48| SELECT | CVD, overweight, no T2D | 2.4 mg weekly | MACE HR 0.80 (95% CI 0.72–0.90) |
+49| FLOW | T2D with CKD | 1.0 mg weekly | Kidney outcome HR 0.76 |
+50
+51The [[STEP trial programme|STEP]] programme established the obesity indication and the [[SUSTAIN trial programme|SUSTAIN]] programme the glycaemic one; [[SELECT trial|SELECT]] and [[FLOW trial|FLOW]] added the outcome indications.{{r|wilding2021,lincoff2023}}
+52
37== References ==53== References ==
38{{reflist}}54{{reflist}}
41<ref name="wilding2021">Wilding JPH, Batterham RL, Calanna S, et al. "Once-weekly semaglutide in adults with overweight or obesity." ''New England Journal of Medicine'' 384(11):989–1002 (2021). DOI:10.1056/NEJMoa2032183. PMID 33567185.</ref>57<ref name="wilding2021">Wilding JPH, Batterham RL, Calanna S, et al. "Once-weekly semaglutide in adults with overweight or obesity." ''New England Journal of Medicine'' 384(11):989–1002 (2021). DOI:10.1056/NEJMoa2032183. PMID 33567185.</ref>
42<ref name="lincoff2023">Lincoff AM, Brown-Frandsen K, Colhoun HM, et al. "Semaglutide and cardiovascular outcomes in obesity without diabetes." ''New England Journal of Medicine'' 389(24):2221–2232 (2023). PMID 37952131.</ref>58<ref name="lincoff2023">Lincoff AM, Brown-Frandsen K, Colhoun HM, et al. "Semaglutide and cardiovascular outcomes in obesity without diabetes." ''New England Journal of Medicine'' 389(24):2221–2232 (2023). PMID 37952131.</ref>
+59
+60== See also ==
+61* [[GLP-1 receptor agonist]]
+62* [[Tirzepatide]]
+63* [[Liraglutide]]
+64* [[Oral semaglutide]]
+65* [[STEP trial programme]]
4366
44{{DEFAULTSORT:Semaglutide}}67{{DEFAULTSORT:Semaglutide}}