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Gastric emptying: difference between revisions

Diff·revision 4 → 5·10:33, 22 Sep 2024

Difference between revision 4 and revision 5 of Gastric emptying. 3 lines changed; the page grew by 373 bytes.

Revision 4 — 00:42, 1 Sep 2024
AnalyticalAnnie (talk)
state that the receptor is a class B G-protein-coupled receptor
2,780 bytes +510
Revision 5 — 10:33, 22 Sep 2024
AmylinAmos (talk)
clarify that concentrations differ substantially between assay platforms
3,153 bytes +373
17Rate is regulated to deliver nutrient to the small intestine at a roughly constant caloric rate, near 2 kcal per minute, regardless of meal composition. The regulator is feedback from nutrient sensing in the duodenum and ileum, mediated by cholecystokinin, [[Glucagon-like peptide-1|GLP-1]], peptide YY and neural reflexes — the so-called ileal brake.{{r|holst2007}}17Rate is regulated to deliver nutrient to the small intestine at a roughly constant caloric rate, near 2 kcal per minute, regardless of meal composition. The regulator is feedback from nutrient sensing in the duodenum and ileum, mediated by cholecystokinin, [[Glucagon-like peptide-1|GLP-1]], peptide YY and neural reflexes — the so-called ileal brake.{{r|holst2007}}
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+19Measurement methods are not interchangeable. Scintigraphy is the reference technique; the ¹³C-octanoate breath test is a validated surrogate; paracetamol absorption is convenient but measures liquid-phase emptying only. A study reporting "delayed emptying" without naming its method is not comparable with one that names a different method.
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19== References ==21== References ==
20{{reflist}}22{{reflist}}
26[[Category:Gastrointestinal physiology]]28[[Category:Gastrointestinal physiology]]
27[[Category:Incretin biology]]29[[Category:Incretin biology]]
+30[[Category:Adverse effects]]
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