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Glucagon-like peptide-1: difference between revisions

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Revision 56 — 12:49, 4 Jul 2025
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78Whether GLP-1 secretion itself is reduced in obesity and type 2 diabetes has been contested for two decades; meta-analytic work suggests differences are small and heterogeneous between studies.{{cn}}78Whether GLP-1 secretion itself is reduced in obesity and type 2 diabetes has been contested for two decades; meta-analytic work suggests differences are small and heterogeneous between studies.{{cn}}
7979
+80== Therapeutic exploitation ==
+81{{main|GLP-1 receptor agonist}}
+82
+83The pathway is targeted clinically in three ways: degradation-resistant peptide agonists ([[exenatide]], [[liraglutide]], [[dulaglutide]], [[semaglutide]]), enzyme inhibition (the DPP-4 inhibitor class), and non-peptide agonists suitable for oral administration ([[orforglipron]]). Multi-receptor constructs extend the approach to GIP ([[tirzepatide]]), glucagon ([[survodutide]], [[retatrutide]]) and [[Amylin receptor agonist|amylin]] ([[CagriSema]]) co-agonism.
+84
80== References ==85== References ==
81{{reflist}}86{{reflist}}
92* Holst JJ. "Discovery of the GLP-1 based drugs for the treatment of diabetes and obesity." ''Peptides'' (2024) — a participant's account of the pathway from isolation to clinic.97* Holst JJ. "Discovery of the GLP-1 based drugs for the treatment of diabetes and obesity." ''Peptides'' (2024) — a participant's account of the pathway from isolation to clinic.
93* Campbell JE, Drucker DJ. "Pharmacology, physiology, and mechanisms of incretin hormone action." ''Cell Metabolism'' 17(6):819–837 (2013).98* Campbell JE, Drucker DJ. "Pharmacology, physiology, and mechanisms of incretin hormone action." ''Cell Metabolism'' 17(6):819–837 (2013).
+99
+100== External links ==
+101* [https://www.ncbi.nlm.nih.gov/gene/2641 GCG — glucagon (gene entry)] — NCBI Gene record for the proglucagon gene.
94102
95== See also ==103== See also ==