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Melanotan II (revision 3)

Old revision·12:22, 1 Dec 2024·ShortDescShai

This is an old revision of this page, as it stood at 12:22, 1 Dec 2024, saved by ShortDescShai with the summary split the pharmacology section from the marketing history. It may differ substantially from the current revision, and any error it contains may since have been corrected.
Melanotan II
ClassMelanocortin receptor agonist
SelectivityNon-selective across MC1R, MC3R, MC4R, MC5R
StatusNot approved in any jurisdiction
Related approved compoundBremelanotide (MC4R-directed)
Compound infobox · conventions

Melanotan II is a synthetic cyclic analogue of α-melanocyte-stimulating hormone and a non-selective agonist at melanocortin receptors. It is not approved in any jurisdiction and is distributed as a research chemical.[1]

Non-selectivity is the defining property. Agonism at MC1R produces melanogenesis, the effect for which it is sought; agonism at MC4R affects appetite and sexual function; agonism at MC3R and MC5R contributes further effects. A single compound producing all of them simultaneously is a pharmacological blunt instrument.[1]

Pharmacology

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The melanocortin system comprises five receptors with distinct distributions. MC1R on melanocytes controls the switch between pheomelanin and eumelanin synthesis; MC4R in the hypothalamus is central to appetite regulation and is the target of approved anti-obesity compounds in rare genetic conditions. See Arcuate nucleus.[2]

Melanotan II activates all of these. Selective compounds developed subsequently — bremelanotide for sexual dysfunction, and MC4R-directed agents for genetic obesity — represent the pharmacological correction of that non-selectivity.[1]

References

  1. ^ a b c Hadley ME, Dorr RT. "Melanocortin peptide therapeutics: historical milestones, clinical studies and commercialization." Peptides 26(10):1687–1689 (2005). PMID 16112778.
  2. ^ Cone RD. "Anatomy and regulation of the central melanocortin system." Nature Neuroscience 8(5):571–578 (2005). PMID 15856065.