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GLP-1 receptor agonist: difference between revisions

Diff·revision 14 → 15·03:14, 20 Nov 2024

Difference between revision 14 and revision 15 of GLP-1 receptor agonist. 13 lines changed; the page grew by 1,583 bytes.

Revision 14 — 17:03, 6 Nov 2024
TriumphThad (talk)
rm the comparison to a compound with no published head-to-head data
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Revision 15 — 03:14, 20 Nov 2024
CiteBot (talk)
bot: expand DOI to full citation
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46Small-molecule agonists such as [[Orforglipron|orforglipron]] achieve oral bioavailability by abandoning the peptide backbone entirely. They are not subject to proteolysis and do not require an absorption enhancer, but they engage a different portion of the receptor and their efficacy relative to injected peptides remains under evaluation.{{r|frias2023}}46Small-molecule agonists such as [[Orforglipron|orforglipron]] achieve oral bioavailability by abandoning the peptide backbone entirely. They are not subject to proteolysis and do not require an absorption enhancer, but they engage a different portion of the receptor and their efficacy relative to injected peptides remains under evaluation.{{r|frias2023}}
4747
+48== Clinical efficacy ==
+49Across the [[STEP trial programme|STEP]], [[SUSTAIN trial programme|SUSTAIN]] and [[SURPASS trial programme|SURPASS]] programmes the class has been assessed against placebo and against active comparators in both diabetic and non-diabetic populations. In people with obesity and without diabetes, weekly semaglutide 2.4 mg produced a mean weight change of −14.9% against −2.4% for placebo at 68 weeks.{{r|wilding2021}} In type 2 diabetes the same class produces glycated-haemoglobin reductions of roughly 1.0–1.8 percentage points, with the larger figures at the higher doses of the more recent agents.{{r|nauck2016}}
+50
+51Cardiovascular outcome trials have shown benefit for several members. The [[SELECT trial|SELECT]] trial reported a reduction in major adverse cardiovascular events in people with established cardiovascular disease and overweight or obesity but without diabetes, and the [[FLOW trial|FLOW]] trial reported slowing of kidney-disease progression.{{r|lincoff2023}} These are outcome findings for specific molecules at specific doses and are not properties of the class as a whole; extrapolation between members is not supported by the trial evidence.
+52
48== References ==53== References ==
49{{reflist}}54{{reflist}}
52<ref name="wilding2021">Wilding JPH, Batterham RL, Calanna S, et al. "Once-weekly semaglutide in adults with overweight or obesity." ''New England Journal of Medicine'' 384(11):989–1002 (2021). DOI:10.1056/NEJMoa2032183. PMID 33567185.</ref>57<ref name="wilding2021">Wilding JPH, Batterham RL, Calanna S, et al. "Once-weekly semaglutide in adults with overweight or obesity." ''New England Journal of Medicine'' 384(11):989–1002 (2021). DOI:10.1056/NEJMoa2032183. PMID 33567185.</ref>
53<ref name="nauck2016">Nauck MA, Meier JJ. "The incretin effect in healthy individuals and those with type 2 diabetes: physiology, pathophysiology, and response to therapeutic interventions." ''The Lancet Diabetes & Endocrinology'' 4(6):525–536 (2016). PMID 26876794.</ref>58<ref name="nauck2016">Nauck MA, Meier JJ. "The incretin effect in healthy individuals and those with type 2 diabetes: physiology, pathophysiology, and response to therapeutic interventions." ''The Lancet Diabetes & Endocrinology'' 4(6):525–536 (2016). PMID 26876794.</ref>
+59<ref name="lincoff2023">Lincoff AM, Brown-Frandsen K, Colhoun HM, et al. "Semaglutide and cardiovascular outcomes in obesity without diabetes." ''New England Journal of Medicine'' 389(24):2221–2232 (2023). DOI:10.1056/NEJMoa2307563. PMID 37952131.</ref>
54<ref name="frias2023">Frías JP, Hsia S, Eyde S, et al. "Efficacy and safety of oral orforglipron in patients with type 2 diabetes: a phase 2 randomised trial." ''The Lancet'' 402(10400):472–483 (2023). PMID 37369232.</ref>60<ref name="frias2023">Frías JP, Hsia S, Eyde S, et al. "Efficacy and safety of oral orforglipron in patients with type 2 diabetes: a phase 2 randomised trial." ''The Lancet'' 402(10400):472–483 (2023). PMID 37369232.</ref>
+61
+62== See also ==
+63* [[Glucagon-like peptide-1]]
+64* [[GLP-1 receptor]]
+65* [[Dual incretin agonist]]
+66* [[Semaglutide]]
+67* [[Liraglutide]]
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56{{DEFAULTSORT:GLP-1 receptor agonist}}69{{DEFAULTSORT:GLP-1 receptor agonist}}